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Showing posts with label skin cancer from tanning. Show all posts
Showing posts with label skin cancer from tanning. Show all posts

Saturday, May 31, 2008

What is skin cancer logoDangers of Tanning

Thursday, May 8, 2008

What is skin cancer logoSunlight effects upon the skin

Ultraviolet radiation (UVR) comprises only about 5% of sunlight, with visible light and infrared radiation making up the remaining 95%. However, it is UVR which is chiefly responsible for the harmful effects of sunlight exposure. On a sunny day, UVA (315-400 nm) accounts for at least 95% of UVR, and UVB (280-315 nm) no more than 5%. UVR has acute and chronic effects on the skin.
Acute effects
Erythema (sunburn)
Sunburn or erythema can vary in intensity from mild redness to oedema, blistering and peeling. Susceptibility to sunburn and tanning depends on the skin phototype of the individual. UVB is much more effective at inducing erythema than UVA. UVB accounts for only about 5% of solar radiation but it contributes to about 80% of the erythema caused by sunlight. Experiments suggest that the synthesis and release of prostaglandin E2 (PGE2), following cyclooxygenase-2 (cox-2) gene activation, and nitric oxide within the dermis are responsible for UV-induced erythema.
Melanogenesis (tanning)
Skin colour is genetically controlled but can be enhanced by UVR exposure. Each skin phototype has a different ability to tan. Tanning and epidermal hyperplasia following UVR exposure protects against erythema and burning, but maintaining this requires repeated exposures, which can result in the chronic effects of photodamage.
Skin phototypes
Skin phototype Response to UVR exposure
I White skin, always burns, never tans (Celtic)
II White skin, burns initially, tans with difficulty
III White skin, rarely burns, tans easily
IV White skin, never burns always tans (Mediterranean)
V Brown skin (Asian)
VI Black skin
Immunosuppression
Both UVA and UVB exposure suppress cutaneous cell-mediated immunity in humans. This effect is significantly higher in skin types I/II than in types III/IV. UVR-induced immunosuppression is thought to play an important role in the emergence of skin cancer, which explains the increased risk of skin cancer in individuals with skin types I/II who have a history of repetitive and intense UVR exposure.
Chronic effects
Photoageing
Photoageing results in dry, deeply wrinkled, inelastic, leathery skin with telangiectasia, mottled pigmentation, freckling and lentigines. Repeated long-term UVR exposure, especially UVB, results in changes within the dermal connective tissue. There is elastosis and degradation and disorganization of collagen fibrils. It is thought that UVR-induced metalloproteinases (endopeptidases that degrade structural proteins) degrade the dermal matrix, which then undergoes imperfect repair.
Photocarcinogenesis
Skin cancer is the long-term result of a complex interaction between UVR exposure and genetics. Genetic factors include skin phototype, DNA repair capacity and immunocompetence. DNA damage occurs following UVR, and this is repaired by nucleotide excision repair. Repeated UV exposure together with suboptimal repair results in the clonal expansion of cells with mutated oncogenes leading to melanoma and non-melanoma skin cancer. Studies have shown that squamous cell carcinomas develop as the consequence of accumulated sun exposure, whereas melanoma and basal cell carcinomas are more dependent on specific patterns of childhood and intermittent high-dose sun exposure. Other important risk factors for the development of skin cancer include UVR-induced immunosuppression as well as immunosuppressant drugs as seen in organ transplant patients.
Photodermatoses
Photodermatoses (diseases caused by sunlight) can be divided into two main categories, those in which the sunlight has a primary role in the condition (primary photosensitivity) and those in which the sunlight acts as an exacerbating factor (secondary photosensitivity).

Saturday, May 3, 2008

What is skin cancer logoTanning and skin cancer facts

There is a popular belief that suntanned skin is healthy and attractive. Many people spend more time in the sun than is good for their skin trying to achieve a fashionable tan.
A considerable percentage of the white skinned population admits to tanning at least once a year either in the sun or in tanning beds.
In a magazine survey, two thirds of teens said they "look better with a tan and feel healthier, more sophisticated," and half said they looked "more athletic".
Tanned skin is thought to be associated with swimming pools, backyard barbecues, dinner parties, and exotic vacations. In this context, tanned skin took on a feature of attractiveness as a signal of being well-traveled, cultured, and supposed evidence of leisure wealth. It also became a signal of health and strength as the bodybuilding and fitness industries increasingly promoted tanning to highlight muscle tone and definition.
Suntan is caused by an increased release of the pigment melanin into the skin's cells after exposure to ultraviolet radiation.
There is convincing evidence from many studies that frequent exposure to any form of UV radiation (solar or artificial) increases the risk for the development of skin cancer.
Preventive measures include avoiding sun exposure between 10 a.m. and 4 p.m, wearing a hat and anti-UV sunglasses which can provide almost 100% protection against ultraviolet radiation entering the eyes and applying a sunscreen that blocks both UVA and UVB rays.

Thursday, February 7, 2008

What is skin cancer logoTop 10 Tips for Preventing Skin Cancer

Skin cancer awareness ---> skin cancer videos ---> Skin cancer ---> How to prevent skin cancer?

Skin cancer doesn't have to happen! Keep watching for 10 tips that will help keep your skin cancer-free.

Monday, November 26, 2007

What is skin cancer logoSkin Cancer -- The Dangers of a Deadly Tan (and Solariums)

Monday, October 29, 2007

What is skin cancer logoClare Oliver


Clare Oliver (25 August 1981 – 13 September 2007) was an Australian woman whose own health crisis prompted her to become an activist, garnering wide media coverage, seeking to ban the use of solariums. Clare's melanoma was first discovered as part of a health check up shortly after she had been employed by SBS television upon completion of a media degree.

She gained publicity on 22 August 2007 by announcing in an open letter that she only had days to live due to melanoma and stating her goal was to reach her 26th birthday. She did, and celebrated at Luna Park in St Kilda, Victoria. Less than three weeks later, she died 8am EST, on 13 September at the Caritas Christi Hospice in Kew.

Clare campaigned in her last days against the lack of regulations governing solariums, however, whilst she believed it a major factor in her illness, she had spent time sun baking at St Kilda in her earlier years, where she grew up.

The Australian government has since made previously voluntary code practices mandatory in the use of tanning beds in Australia.The Victorian government also announced it would enact legislation to tighten the control of solariums by the end of 2007.

Clare is survived by her mother, Priscilla Lau Oliver.

Legacy

Clare Oliver's Legacy is her vehement disapproval of Solarium's use in communities. In her life she campaigned for their complete ban and her legacy pertains the the dangers of sun tanning in general and how destructive skin cancers can be to anyone's life.

From Wikipedia, the free encyclopedia

Sunday, October 21, 2007

What is skin cancer logoClare Oliver

Saturday, October 20, 2007

What is skin cancer logoClare Oliver